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"Our findings suggest that macrophages from the lungs of patients with COPD have a defect in a process called phagocytosis involved in the uptake of bacteria. We discovered that activation of the Nrf2 pathway induced by sulforaphane restored the ability of lung macrophages to bind and take up bacteria," said Shyam Biswal, PhD, professor in the Bloomberg School's Department of Environmental Health Sciences and senior author of the study. "The study provides proof of concept that activating the Nrf2 pathway can restore the ability of macrophage to phagocytose, or bind with bacteria, and clear it from the lungs of patients with COPD."

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